![]() ![]() Qingwen Qian 1, Mark Li 1,2, zeyuan Zhang 1, Shannon Davis 3, Kamal Rahmouni 4, Andrew W Norris 5, Wen-Xing Ding 6, Huojun Cao 7 and Ling Yang 1, (1)Foe Diabetes Center, Department of Anatomy and Cell Biology, University of Iowa, (2)University of Iowa, (3)University of South Carolina, (4)Department of Neuroscience and Pharmacology, University of Iowa, (5)Department of Internal Medicine, University of Iowa, (6)Pharmacology, Toxicology and Therapeutics, University of Kansas Medical Center, (7)Iowa Institute for Oral Health Research, University of Iowa College of Dentistryīackground: The hepatic immuno-metabolic response is tightly controlled by hormones released from pituitary endocrine cells that rely on an intact endoplasmic reticulum (ER) function. ![]() ![]() DISRUPTION OF PITUITARY-LIVER UPR INTERACTION CONTRIBUTES TO NAFLD ![]()
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